Dewi Nurul Mustaqimah
Department of Periodontics, Faculty of Dentistry, Universitas Indonesia, Jakarta 10430

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Analisis Antibodi IgG Spesifik Fimbria Porphyromonas gingivalis dengan Metoda Elisa pada Penderita Early-Onset Periodontitis Mustaqimah, Dewi Nurul; Sukardi, Irene; Masahiro, Yoneda; Maeda, K.
Journal of Dentistry Indonesia Vol 7, No 2 (2000): August
Publisher : Faculty of Dentistry, University of Indonesia

Show Abstract | Download Original | Original Source | Check in Google Scholar | Full PDF (458.679 KB) | DOI: 10.14693/jdi.v20i3.684

Abstract

The adherence of bacteria to the host tissues is a first step in the development of infections. Bacterial fimbriae have been shown to play an important role in the interaction between bacteria and host cells or among bacterial cells. Optical density of serum IgG specific Porphyromonas gingivalis strain ATCC 33277 fimbriae antibodies has been estimated in 32 cases of Early-Onset Periodontitis and 14 healthy periodontal subjects as control. The mean level of IgG were significantly elevated (p=0,000) in their cases. The data support the role of P. gingivalis as a key pathogen in Early-Onset Periodontitis.
Kondisi Subpopulasi Limfosit Penderita Early-Onset Periodontitis Pengunjung Klinik Periodonsia Rumah Sakit Gigi & Mulut FKG UI Mustaqimah, Dewi Nurul
Journal of Dentistry Indonesia Vol 7, No 2 (2000): August
Publisher : Faculty of Dentistry, University of Indonesia

Show Abstract | Download Original | Original Source | Check in Google Scholar | Full PDF (579.69 KB) | DOI: 10.14693/jdi.v20i3.685

Abstract

In the last decade, much attention has been devoted to study immunologic factors in early-onset periodontitis (EOP). This study was designed to investigate peripheral blood lymphocyte subpopulations cells in patients with EOP, 32 patients with EOP and 10 normal healthy control subjects were included in the study. Peripheral blood T-lymphocytes, helper T-cells, suppressor T-cells, B-lymphocytes, natural killer (NK) cells and the Th/Ts ratio were determined using appropriate monoclonal antibodies and the indirect immunofluorescence method. In the EOP group, T-lymphocytes and helper T-cells were found to be significantly lowere where as NK cells was significantly higher than the control group. They had normal numbers of B-lymphocytes, suppressor T-cells, and the Th/Ts ratio. These findings could contribute to the science about EOP in Indonesia, and to the immunopathogenesis of EOP.
Masalah Nyeri pada Kasus Penyakit Periodontal dan Cara Mengatasinya Mustaqimah, Dewi Nurul
Journal of Dentistry Indonesia Vol 9, No 2 (2002): August
Publisher : Faculty of Dentistry, University of Indonesia

Show Abstract | Download Original | Original Source | Check in Google Scholar | Full PDF (2201.788 KB) | DOI: 10.14693/jdi.v9i2.675

Abstract

Pain in periodontal disease has many variations from slight discomfort to very severe pain. Pain can be a tormenting experience for some dental patients. Every patient wants to avoid pain and this is a task for the dentists effort in clinical practice. Treatment of pain can be vary in periodontal disease, but the different optional treatments oral physiotherapy is one to be recommended. The patient should be well motivated to wards good oral hygiene, to relieve pain and prevent its recurrence.
Zat Kimia Berbentuk Gas yang Dapat Mengawali Pengrusakan Jaringan Periodonsium Mustaqimah, Dewi Nurul
Journal of Dentistry Indonesia Vol 9, No 3 (2002): December
Publisher : Faculty of Dentistry, University of Indonesia

Show Abstract | Download Original | Original Source | Check in Google Scholar | Full PDF (1861.998 KB) | DOI: 10.14693/jdi.v9i3.718

Abstract

The etiology of periodontal inflammation are periodontal pathogenic bacteriae and defence immune responses, as well as others local and systemic factors. Several predisposing factors, however, play an important role and unfortunately are less appreciated either by the dentist or the patient himself. Among these is the volatile sulphur compounds (VSC), which is found to be harmfull to the cell by causing the degradation of proteoglycan and glycoprotein, and result in the increase of the cell premeability. The volatile sulphuric acid-risk exposed-workers from galvanizing or battery factories have significantly greater portion of deep pockets than the non-risk exposed-one. Cigarettes smoke also known to contain more than 4,000 toxic agents which could delay the healing process of periodontal surgery lesions. It could be concluded that the volatile chemical compounds should be appreciated to lessen the morbidity rate of periodontal diseases.
The Effect of Daily Lifestyle on Periodontal Health Mustaqimah, Dewi Nurul; Darmabrata, Wahyadi; Safrina, Hayatun; Sukardi, Irene
Journal of Dentistry Indonesia Vol 12, No 2 (2005): August
Publisher : Faculty of Dentistry, University of Indonesia

Show Abstract | Download Original | Original Source | Check in Google Scholar | Full PDF (3595.516 KB) | DOI: 10.14693/jdi.v12i2.865

Abstract

The environments to which individuals have been exposed throughout their lives from birth to the present time have comprised the individual and community lifestyles. Lifestyles are known to be associated with physical health status. Physical health and periodontal status impacts on the life quality.Periodontal disease is generally considered to be resulted from an imbalance between potentially pathogenic microbes and the nature and efficacy of local and systemic host responses. Factors related to social environments such as poor lifestyles and negative psycosocial conditions may also be important in disease development. Good health practices are associated with better mental health status, as well as with periodontal health status.
Terapi periodontal non-bedah Non-surgical periodontal therapy Zulfa, Liana; Mustaqimah, Dewi Nurul
Journal of Dentomaxillofacial Science Vol 10, No 1 (2011): Formerly Jurnal Dentofasial ISSN 1412-8926
Publisher : DiscoverSys Inc

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.15562/jdmfs.v10i1.250

Abstract

Non surgical periodontal therapy is the first step in periodontal therapy procedure. Elimination of biofilm andmineral deposit from tooth surface is a basic of periodontal therapy. Clinical data showed that the success of longterm periodontal therapy depends on the result of first step compare to spesific surgical therapy. Methods used innon surgical therapy are mechanical instrumentation, ultrasonic debridement, supragingival and subgingivalirigation, localized drugs, systemic antibiotics, and host response modulation. The successful of periodontal therapydepends on the management of negative environment and reduction of bacterial pathogen through a combination ofbetter environment which is less anaerobic for good microorganism to live. The aim of non surgical therapy is toreduce microbial etiology and factors involved in the progression of gingiva and periodontal. The end result is tostop the disease progression and to gain healthy gingiva and tissue condition.
THE ETIO-PATHOGENESIS OF PERIODONTAL DISEASE Mustaqimah, Dewi Nurul
Indonesian Journal of Tropical and Infectious Disease Vol 1, No 3 (2010)
Publisher : Institute of Topical Disease

Show Abstract | Download Original | Original Source | Check in Google Scholar | Full PDF (407.998 KB) | DOI: 10.20473/ijtid.v1i3.2196

Abstract

The etiology of polymicrobial disease such as periodontitis is likely to be more complex than suggested by the traditional paradigm of disease involving a single virulent organism which up to now has been believed. This review limits its discussion to the other subgingival microbiota which is not yet cultivable, however it is suggested be implicated with the severity of periodontal disease. The intricate interactions between viruses and bacteria within periodontal pockets as a co-infection process reveal its role in the etio-pathogenesis of periodontal disease. Also Archaea domain participate in syntrophic relationship with the microbiota life members in the subgingival crevice, promote colonization by special bacterial group during periodontitis. It is clear that periodontal diseases are not monoinfections.